Dzmitry, я не знаю, насколько это обнадеживает. У меня средняя (по все анализам) мочевина за эти полтора года = 20.3 ммоль/л. У меня ощущение (и подозрение), что это не самый лучший вариант. Склонен согласиться с табличкой в FAQ на сайте, где написано, что оценкой скорости прогрессирования надо заниматься на уровне СКФ=60-89 мл/мин, а на моей стадии (30 мл/мин) надо заниматься Оценкой и лечением осложнений и подготовкой к ЗПТ. Потому что чувствую я себя большую часть времени отвратительно. Вот снизить бы мочевину до 10..

Erratum in:
J Am Soc Nephrol 1995 Oct;6(4):1318.

Dietary protein restriction, blood pressure control, and the progression of polycystic kidney disease. Modification of Diet in Renal Disease Study Group.
Klahr S, Breyer JA, Beck GJ, Dennis VW, Hartman JA, Roth D, Steinman TI, Wang SR, Yamamoto ME.

National Institute of Diabetes, Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD, USA.

In the Modification of Diet in Renal Disease Study, a follow-up (mean, 2.2 yr) of 200 study participants with autosomal dominant polycystic kidney disease (ADPKD) was conducted to determine the effect of lowering protein intake and blood pressure on the rate of decline in GFR. The rate of decline was faster in participants with ADPKD than in persons with other diagnoses, reflecting, in part, faster disease progression in the ADPKD group. Baseline characteristics that predicted a faster rate of decline in GFR in persons with ADPKD were greater serum creatinine (independent of GFR), greater urinary protein excretion, higher mean arterial pressure (MAP), and younger age. In patients with initial GFR values between 25 and 55 mL/min per 1.73 m2, neither assignment to a low-protein diet group nor assignment to a low blood pressure group significantly reduced the rate of decline of GFR in ADPKD participants. Similarly, the decline in GFR was not related to achieved protein intake or MAP. In participants with GFR values between 13 and 24 mL/min per 1.73 m2, assignment to the low MAP group led to a somewhat more rapid decline in GFR. However, the more rapid decline in GFR did not appear to be due to a detrimental effect of low blood pressure or the antihypertensive agents used to reach the low blood pressure goal. Lower protein intake, but not prescription of the keto acid-amino acid supplement, was marginally associated with a slower progression of renal disease.

Было бы интересно прочитать и эту статью, но, к сожалению, абстракт неполон.
Nephron. 1993;64(2):207-15.
Effect of changes in daily protein intake on renal function in chronic renal insufficiency: differences in reaction according to disease entity.
Schaap GH, Bilo HJ, van der Meulen J, Oe PL, Donker AJ.

А вот здесь, в статье которую я пыжусь сейчас прочесть ("Dietary Protein Restriction, Blood Pressure control and the progression of Polycystic Kidney Didease"), написано:
"...participants had their GFR determined by the clearance of iothalamate ... their protein intake estimated by 24-h urine urea nitrogen excretion ... and by 3-day food records ...."
(мой примерный перевод: СКФ участников (исследования) определяли по клиренсу iothalamate... (их) потребление белка оценивалось по 24-часовой экскреции с мочой азота мочевины.. и по 3-х дневным пищевым дневникам...)